The Neuroplasticity Program will focus on recovery approaches that modify synapses and circuits to enhance functional capacity. Neuroplasticity strategies will attempt to maximize function from existing cells as a critical component of promoting recovery from traumatic brain injury (TBI). Neuroplasticity studies will assist the development of neuroregenerative strategies since newly generated cells must establish the appropriate neuronal-glial interactions and/or integrate into the appropriate circuitry to contribute to the recovery of function.
Neuroplasticity in human sensory cortex
Functional Maps of Neuronal and Glial Activity Following TBI, Stress and TBI
Motor learning after TBI
Epigenetic factors and cortical map plasticity in the model of TBI
Executive Function & Social Cognitive Adaptive Neuroplasticity
Differential Dysregulation of Subcortical Synaptic Plasticity after TBI
Does TBI alter extrinsic and intrinsic connectivity in sensory cortex?
Development of an organotypic brain slice model for shear injury
The role of reward in human neuroplasticity
Retrograde injury signaling and cytoskeletal alterations in barrel cortex after TBI
NR2B-selective antagonist to ameliorate posttraumatic epileptogenesis and its associated comorbidities
Improving rehabilitation of Decision Making deficits after TBI
Does training influence cortical remodeling after TBI?
Monitoring cognitive function in the prefrontal cortex functional near-infrared spectroscopy (fNIRS)
Balance between Inhibition and Excitation and the Vascular Trauma in Sensory Barrel Cortex after TBI
Contributions of Cost and Reward Response to Fatigue
Propagated Deep Brain Neuronal Plasticity after Prefrontal Cortical Injury